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Both classes exhibit pulmonary protective effects via systemic anti-inflammatory reprogramming, with clinical studies demonstrating reduced exacerbation risk in T2DM patients with comorbid COPD ( As previously discussed, metabolic imbalance and mitochondrial dysfunction under diabetic conditions are key mechanisms leading to lung injury
5 Efforts were made to minimize the number of animals used
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Studies have demonstrated that induction of HO-1 expression in macrophages yields amplified antioxidant efficacy and mitigates the inflammatory aspect of atherosclerotic lesions
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